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Traumatic Brain Injury (TBI)

  • Cannabidiol, a nonpsychoactive Cannabis constituent, protects against myocardial ischemic reperfusion injury
  • Cannabidiol prevents infarction via the non-CB1 cannabinoid receptor mechanism
  • Cannabidiol prevents cerebral infarction via a serotonergic 5-hydroxytryptamine1A receptor-dependent mechanism
  • Cannabidiol administration after hypoxia-ischemia to newborn rats reduces long-term brain injury and restores neurobehavioral function 
  • Protective Effects of Cannabidiol Against Hippocampal Cell Death and Cognitive Impairment Induced by Bilateral Common Carotid Artery Occlusion in Mice
  • Repeated treatment with cannabidiol but not Delta9-tetrahydrocannabinol has a neuroprotective effect without the development of tolerance
  • CBD reduces brain damage and improves functional recovery in a neonatal rat model of arterial ischemic stroke
  • Acute upregulation of neuronal mitochondrial type-1 cannabinoid receptor and it’s role in metabolic defects and neuronal apoptosis after TBI
  • An endogenous cannabinoid (2-AG) is neuroprotective after brain injury
  • Cannabinoid agonist rescues learning and memory after a traumatic brain injury
  • Early increase of cannabinoid receptor density after experimental traumatic brain injury in the newborn piglet
  • Endocannabinoids and traumatic brain injury
  • Preclinical evaluation of SMM-189, a cannabinoid receptor 2-specific inverse agonist
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Disclaimer: The statements made regarding these products have not been evaluated by the Food and Drug Administration. The efficacy of these products has not been confirmed by FDA-approved research. These products are not intended to diagnose, treat, cure or prevent any disease. All information presented here is not meant as a substitute for or alternative to information from health care practitioners. Please consult your health care professional about potential interactions or other possible complications before using any product. The Federal Food, Drug and Cosmetic Act requires this notice.

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